Litcius/Paper detail

Sub-Acute Treatment of Curcumin Derivative J147 Ameliorates Depression-Like Behavior Through 5-HT1A-Mediated cAMP Signaling

Jianxin Li, Ling Chen, Gaowen Li, Xiaojuan Chen, Sisi Hu, Liang Zheng, Victor Luria, Jinpeng Lv, Yindi Sun, Ying Xu, Yingcong Yu

2020Frontiers in Neuroscience27 citationsDOIOpen Access PDF

Abstract

BACKGROUND: receptors and downstream cAMP-BDNF signaling. METHODS: agonist or its antagonist was used to determine which 5-HT receptor subtype is involved in the antidepressant-like effects of J147. The downstream signaling molecules such as cAMP, PKA, pCREB, and BDNF were also measured to determine the mechanism of action. RESULTS: receptor antagonist NAS-181 did not appreciably alter the effects of J147 on depression-like behaviors. Moreover, pretreatment with NAD-299 blocked J147-induced increases in cAMP, PKA, pCREB, and BDNF expression in the hippocampus, while 8-OH-DPAT enhanced the effects of J147 on these proteins' expression. CONCLUSION: -dependent cAMP/PKA/pCREB/BDNF signaling.

Topics & Concepts

ReceptorAgonistSerotoninAntidepressantBehavioural despair testInternal medicineMonoamine neurotransmitterEndocrinology5-HT receptorCurcuminPharmacologyBrain-derived neurotrophic factor5-HT1A receptorReceptor antagonistHippocampusAntagonistNeurotrophic factorsMedicineNerve injury and regenerationNeuroscience and Neuropharmacology ResearchNeuropeptides and Animal Physiology
Sub-Acute Treatment of Curcumin Derivative J147 Ameliorates Depression-Like Behavior Through 5-HT1A-Mediated cAMP Signaling | Litcius