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Pseudorabies Virus Inhibits Type I and Type III Interferon-Induced Signaling via Proteasomal Degradation of Janus Kinases

Yue Yin, Nicolás Romero, Herman W. Favoreel

2021Journal of Virology33 citationsDOIOpen Access PDF

Abstract

Type I and III interferons (IFNs) trigger signaling via Janus kinases that phosphorylate and activate signal transducer and activator of transcription (STAT) transcription factors, leading to the expression of antiviral interferon-stimulated genes (ISGs) that result in an antiviral state of host cells. Viruses have evolved various mechanisms to evade this response. Our results indicate that an alphaherpesvirus, the porcine pseudorabies virus (PRV), inhibits both type I and III IFN signaling pathways by triggering proteasome-dependent degradation of the key Janus kinases Jak1 and Tyk2 and consequent inhibition of STAT1 phosphorylation and suppression of ISG expression. Moreover, we found that this inhibition is not caused by incoming virions and does not depend on expression of the viral EP0 protein or viral true late proteins. These data for the first time address alphaherpesvirus evasion of type III IFN-mediated signaling and reveal a previously uncharacterized alphaherpesvirus mechanism of IFN evasion via proteasomal degradation of Janus kinases.

Topics & Concepts

Janus kinaseBiologySTAT proteinSTAT1InterferonPhosphorylationKinaseSignal transductionCell biologyTyrosine kinase 2JAK-STAT signaling pathwayJanus kinase 1PseudorabiesVirologyVirusTyrosine kinaseSTAT3GeneticsReceptorPlatelet-derived growth factor receptorGrowth factorHerpesvirus Infections and TreatmentsViral Infections and Immunology Researchinterferon and immune responses
Pseudorabies Virus Inhibits Type I and Type III Interferon-Induced Signaling via Proteasomal Degradation of Janus Kinases | Litcius