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IRE1α overexpression in malignant cells limits tumor progression by inducing an anti-cancer immune response

Adriana Martinez-Turtos, Rachel Paul, Manuel Grima-Reyes, Hussein Issaoui, Adrien Krug, Rana Mhaidly, Jozef P. Bossowski, Johanna Chiche, Sandrine Marchetti, Els Verhoeyen, Éric Chevet, Jean‐Ehrland Ricci

2022OncoImmunology21 citationsDOIOpen Access PDF

Abstract

tumor-suppressive effect of overexpressing IRE1α in tumor cells was associated with IRE1α RNAse activity driving both XBP1 mRNA splicing and regulated IRE1-dependent decay of RNA (RIDD). We showed that the tumor-suppressive phenotype upon IRE1α overexpression was characterized by the induction of apoptosis in tumor cells along with an enhanced adaptive anti-cancer immunosurveillance. Hence, our work indicates that IRE1α overexpression and/or activation in tumor cells can limit tumor growth in immunocompetent mice. This finding might point toward the need of adjusting the use of IRE1α inhibitors in cancer treatments based on the predominant outcome of the RNAse activity of IRE1α.

Topics & Concepts

Unfolded protein responseCancer researchXBP1Endoplasmic reticulumLewis lung carcinomaCancerBiologyImmune systemTumor progressionRNA splicingImmunologyMetastasisCell biologyRNAGeneBiochemistryGeneticsEndoplasmic Reticulum Stress and DiseaseAutophagy in Disease and TherapyIron Metabolism and Disorders
IRE1α overexpression in malignant cells limits tumor progression by inducing an anti-cancer immune response | Litcius