Cardiomyocytes recruit monocytes upon SARS-CoV-2 infection by secreting CCL2
Liuliu Yang, Benjamin E. Nilsson-Payant, Yuling Han, Fabrice Jaffré, Jiajun Zhu, Pengfei Wang, Tuo Zhang, David Redmond, Sean Houghton, Rasmus Møller, Daisy A. Hoagland, Lucía Carrau, Shu Horiuchi, Marisa Goff, Jean K. Lim, Yaron Bram, Chanel Richardson, Vasuretha Chandar, Alain Borczuk, Yaoxing Huang, Jenny Xiang, David D. Ho, Robert E. Schwartz, Benjamin R. tenOever, T.E. Evans, Shuibing Chen
Abstract
(Stem Cell Reports 16, 2274–2288; September 14, 2021) In the experimental procedures of the main text and in the supplemental information, the authors have now corrected the source of Golden Syrian hamsters to “Charles River” instead of “Jackson Laboratory” (the latter of which mistakenly appeared in the original version). The authors apologize for the confusion caused. Cardiomyocytes recruit monocytes upon SARS-CoV-2 infection by secreting CCL2Yang et al.Stem Cell ReportsJuly 19, 2021In BriefIn this article, Dr. Shuibing Chen and colleagues report the direct evidence of SARS-CoV-2 infection of cardiomyocytes in vivo and establish an in vitro model to study immune cell infiltration. They suggest that SARS-CoV-2 infection leads to secretion of CCL2, which recruits monocyte migration. Full-Text PDF Open Access