Acute hypokinetic-rigid syndrome following SARS-CoV-2 infection
Antonio Méndez-Guerrero, María Isabel Laespada-García, Adolfo Gómez‐Grande, Mariano Ruiz-Ortiz, Víctor Antonio Blanco-Palmero, Francisco Javier Azcárate-Díaz, Pablo Rábano‐Suárez, Eva Torres, Carlos Pablo de Fuenmayor‐Fernández de la Hoz, Diana Vega Pérez, Raquel Rodríguez-Montalbán, Alfredo Pérez‐Rivilla, Javier Sayas Catalán, Ana Ramos‐González, Jesús González de la Aleja
Abstract
OBJECTIVE: To report a case of a patient infected with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) who acutely developed a hypokinetic-rigid syndrome. METHODS: score values of the striatum. RESULTS: We report a previously healthy 58-year-old man who developed hyposmia, generalized myoclonus, fluctuating and transient changes in level of consciousness, opsoclonus, and an asymmetric hypokinetic-rigid syndrome with ocular abnormalities after a severe SARS-CoV-2 infection. DaT-SPECT confirmed a bilateral decrease in presynaptic dopamine uptake asymmetrically involving both putamina. Significant improvement in the parkinsonian symptoms was observed without any specific treatment. CONCLUSION: This case study provides clinical and functional neuroimaging evidence to support that SARS-CoV-2 can gain access to the CNS, affecting midbrain structures and leading to neurologic signs and symptoms.