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MicroRNA-375 exacerbates knee osteoarthritis through repressing chondrocyte autophagy by targeting ATG2B

Hongxing Li, Zhi‐Ling Li, Yigang Pi, Yang Chen, Lin Mei, Yong Luo, Jingping Xie, Xinzhan Mao

2020Aging57 citationsDOIOpen Access PDF

Abstract

OBJECTIVE: This study aimed to explore the underlying mechanism of miR-375 in exacerbating osteoarthritis (OA). RESULTS: results further verified that inhibition of miR-375 could relieve OA-related symptoms. CONCLUSION: miR-375 can inhibit the expression of ATG2B in chondrocytes, suppress autophagy and promote the ERs. It suggests that miR-375 could be considered to be a key therapy target for OA. METHODS: , OA mice model was established, HE and Safranin O and Fast Green staining, as well as the OARSI and modified Mankin scores, were applied to measure the OA cartilage damage severity.

Topics & Concepts

ChondrocyteAutophagymicroRNAOsteoarthritisWestern blotIn vivoDownregulation and upregulationCartilageApoptosisEndoplasmic reticulumFlow cytometryCell biologyTransfectionChemistryCancer researchBiologyMolecular biologyMedicinePathologyGeneBiochemistryAnatomyAlternative medicineBiotechnologyOsteoarthritis Treatment and MechanismsCancer-related molecular mechanisms researchExtracellular vesicles in disease
MicroRNA-375 exacerbates knee osteoarthritis through repressing chondrocyte autophagy by targeting ATG2B | Litcius