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Increased Axon Initial Segment Length Results in Increased Na+ Currents in Spinal Motoneurones at Symptom Onset in the G127X SOD1 Mouse Model of Amyotrophic Lateral Sclerosis

H.S. Jørgensen, Dennis Bo Jensen, K. P. Dimintiyanova, V.S. Bonnevie, Anne Hedegaard, Janna Lehnhoff, Mihai Moldovan, L. Grøndahl, Claire Francesca Meehan

2020Neuroscience34 citationsDOIOpen Access PDF

Topics & Concepts

Amyotrophic lateral sclerosisSOD1NeuroscienceAxonAntidromicGenetically modified mouseIn vivoSpinal cordMedicineElectrophysiologyPathologyBiologyTransgeneDiseaseBiotechnologyBiochemistryGeneAmyotrophic Lateral Sclerosis ResearchNeurogenetic and Muscular Disorders ResearchParkinson's Disease Mechanisms and Treatments
Increased Axon Initial Segment Length Results in Increased Na+ Currents in Spinal Motoneurones at Symptom Onset in the G127X SOD1 Mouse Model of Amyotrophic Lateral Sclerosis | Litcius