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Pharmacological Targeting of Vacuolar H+-ATPase via Subunit V1G Combats Multidrug-Resistant Cancer

Yuezhou Wang, Lei Zhang, Yanling Wei, Wei Huang, Li Li, Anan Wu, Anahita Dastur, Patricia Greninger, Walter M. Bray, Chen‐Song Zhang, Mengqi Li, Wenhua Lian, Zhiyu Hu, Xiaoyong Wang, Gang Liu, Luming Yao, Jih‐Hwa Guh, Lanfen Chen, Hongrui Wang, Dawang Zhou, Sheng‐Cai Lin, Qingyan Xu, Yuemao Shen, Jianming Zhang, Melissa S. Jurica, Cyril H. Benes, Xianming Deng

2020Cell chemical biology27 citationsDOIOpen Access PDF

Topics & Concepts

mTORC1Multiple drug resistanceNatural productProtein subunitCell growthCytotoxicityBiologyATPaseCancer cellCancer researchChemistryPharmacologyCancerIn vitroDrug resistanceSignal transductionBiochemistryPI3K/AKT/mTOR pathwayEnzymeGeneGeneticsMicrobiologyATP Synthase and ATPases ResearchDrug Transport and Resistance MechanismsRNA and protein synthesis mechanisms
Pharmacological Targeting of Vacuolar H+-ATPase via Subunit V1G Combats Multidrug-Resistant Cancer | Litcius