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CCL20/CCR6 chemokine signaling is not essential for pathogenesis in an experimental autoimmune encephalomyelitis mouse model of multiple sclerosis

Nozomi Sachi, Naganori Kamiyama, Benjawan Saechue, Sotaro Ozaka, Astri Dewayani, Shimpei Ariki, Thanyakorn Chalalai, Yasuhiro Soga, Chiaki Fukuda, Yomei Kagoshima, Supanuch Ekronarongchai, Takashi Kobayashi

2022Biochemical and Biophysical Research Communications13 citationsDOIOpen Access PDF

Topics & Concepts

Experimental autoimmune encephalomyelitisC-C chemokine receptor type 6CCL20ImmunologyImmune systemChemokineBiologyChemokine receptorMultiple sclerosisEncephalomyelitisAutoimmune diseaseT cellAntibodyT-cell and B-cell ImmunologyImmunotherapy and Immune ResponsesChemokine receptors and signaling
CCL20/CCR6 chemokine signaling is not essential for pathogenesis in an experimental autoimmune encephalomyelitis mouse model of multiple sclerosis | Litcius