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IRAK-M Ablation Promotes Status Epilepticus-Induced Neuroinflammation via Activating M1 Microglia and Impairing Excitatory Synaptic Function

Xiao-Shan Liang, Tinglin Qian, Yifan Xiong, Xiaotao Liang, Yuewen Ding, Xiaoyu Zhu, Yun-Lv Li, Jie-Li Zhou, Leyi Tan, Weipeng Li, Wei Xie

2023Molecular Neurobiology17 citationsDOI

Topics & Concepts

NeuroinflammationEpileptogenesisMicrogliaHippocampal formationNeuroscienceGlutamatergicStatus epilepticusBiologyEpilepsyCell biologyGlutamate receptorImmunologyReceptorInflammationBiochemistryNeuroinflammation and Neurodegeneration MechanismsImmune Response and InflammationNeuroscience and Neuropharmacology Research
IRAK-M Ablation Promotes Status Epilepticus-Induced Neuroinflammation via Activating M1 Microglia and Impairing Excitatory Synaptic Function | Litcius