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Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage

Tiago J. Costa, Simone R. Potje, Thais Fernanda de Campos Fraga‐Silva, Júlio Alves da Silva-Neto, Paula R. Barros, Daniel Rodrigues, Mirele R. Machado, Ronaldo B. Martins, Rosangela Aparecida dos Santos Eichler, Maíra Nilson Benatti, Keyla Santos Guedes de Sá, Carlos Eduardo L. Almado, Ítalo A. Castro, Marjorie Cornejo Pontelli, Leonardo La Serra, Fernando S. Carneiro, Christiane Becari, Paulo Louzada‐Júnior, Renê Donizeti Ribeiro de Oliveira, Dario S. Zamboni, Eurico Arruda, Maria Auxiliadora‐Martins, Fernanda R. Giachini, Vânia Luiza Deperon Bonato, Natasha E. Zachara, Gisele Facholi Bomfim, Rita C. Tostes

2021Vascular Pharmacology122 citationsDOIOpen Access PDF

Topics & Concepts

Mitochondrial DNATLR9Cell biologySevere acute respiratory syndrome coronavirus 2 (SARS-CoV-2)ChemistryCoronavirus disease 2019 (COVID-19)BiologyGeneticsMedicineGeneDNA methylationGene expressionDiseaseInfectious disease (medical specialty)PathologyCOVID-19 Clinical Research StudiesLong-Term Effects of COVID-19Mitochondrial Function and Pathology
Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage | Litcius