Litcius/Paper detail

Taurochenodeoxycholic acid activates autophagy and suppresses inflammatory responses in microglia of MPTP-induced Parkinson’s disease mice via AMPK/mTOR, AKT/NFκB and Pink1/Parkin signaling pathways mediated by Takeda G protein-coupled receptor 5

Chenyang Ni, Lupeng Wang, Yuyan Bai, Fei Huang, Hailian Shi, Hui Wu, Xiaojun Wu, Jin Huang, Jin Huang

2025Free Radical Biology and Medicine16 citationsDOI

Topics & Concepts

PINK1ParkinPI3K/AKT/mTOR pathwayAutophagyMicrogliaAMPKProtein kinase BMPTPCancer researchCell biologyParkinson's diseaseMedicineNeuroscienceChemistrySignal transductionInflammationKinaseDiseaseImmunologyBiologyInternal medicineProtein kinase AApoptosisBiochemistryAdenosine and Purinergic SignalingParkinson's Disease Mechanisms and TreatmentsNeuroinflammation and Neurodegeneration Mechanisms
Taurochenodeoxycholic acid activates autophagy and suppresses inflammatory responses in microglia of MPTP-induced Parkinson’s disease mice via AMPK/mTOR, AKT/NFκB and Pink1/Parkin signaling pathways mediated by Takeda G protein-coupled receptor 5 | Litcius