Loss of Cxcr4 in Endometriosis Reduces Proliferation and Lesion Number while Increasing Intraepithelial Lymphocyte Infiltration
Aya Tal, Reshef Tal, Harvey J. Kliman, Hugh S. Taylor
Topics & Concepts
EndometriosisLesionBiologyConditional gene knockoutCXCR4PathologyKnockout mouseImmune systemMedicineImmunologyReceptorChemokinePhenotypeGeneBiochemistryEndometriosis Research and TreatmentReproductive System and PregnancyOvarian cancer diagnosis and treatment