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H3K18 lactylation drives the progression of silica nanoparticles-induced pulmonary fibrosis via promoting macrophage M1 polarization

Lili Shi, Lin Zhang, Zhenfang Ma, Zhaohui Hu, Zihan Geng, Hao Liu, Jingyuan Wang, Ke Feng, Ke Feng, Yan Liu, Cun‐Hui Zhang, Y. G. Xie, Mengqi Zou, Qingping Liu, Ze Yang, Yaxian Pang, Yujie Niu, Lei Bao, Rong Zhang, Lei Bao, Rong Zhang

2025Journal of Hazardous Materials21 citationsDOI

Topics & Concepts

Pulmonary fibrosisMacrophage polarizationNanoparticleFibrosisMacrophagePolarization (electrochemistry)ChemistryCancer researchCell biologyBiophysicsMaterials scienceMedicineNanotechnologyBiochemistryPathologyBiologyIn vitroPhysical chemistryInterstitial Lung Diseases and Idiopathic Pulmonary FibrosisOccupational and environmental lung diseasesMacrophage Migration Inhibitory Factor
H3K18 lactylation drives the progression of silica nanoparticles-induced pulmonary fibrosis via promoting macrophage M1 polarization | Litcius