The hepatic compensatory response to elevated systemic sulfide promotes diabetes
Roderick N. Carter, Matthew Gibbins, Martin E. Barrios‐Llerena, Stephen E. Wilkie, Lydia Freddolino, Marouane Libiad, Victor Vitvitsky, Barry Emerson, Thierry Le Bihan, Madara Brice, Huizhong Su, Scott G Denham, Natalie Homer, Clare Mc Fadden, Anne Tailleux, Nourdine Faresse, Thierry Sulpice, François Briand, Tom Gillingwater, Kyo Han Ahn, Subhankar Singha, Claire McMaster, Richard C. Hartley, Bart Staels, Gillian A. Gray, Andrew J. Finch, Colin Selman, Ruma Banerjee, Nicholas M. Morton
Abstract
mice. We reveal a critical role of TST in hepatic metabolism that has implications for sulfide donor strategies in the context of metabolic disease.
Topics & Concepts
SulfideLipid metabolismGluconeogenesisEndocrinologyInternal medicineMetabolismChemistryBeta oxidationContext (archaeology)BiochemistryBiologyMedicineOrganic chemistryPaleontologySulfur Compounds in BiologyAlcohol Consumption and Health EffectsLiver Disease Diagnosis and Treatment