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Re-evaluating CD57 as a marker of T cell senescence: implications for immune ageing and differentiation

Gaëlle Autaa, Daniil Korenkov, Josine van Beek, Isabelle Pellegrin, Béatrice Parfait, Debbie van Baarle, Odile Launay, Éric Tartour, Victor Appay

2025Immunity & Ageing7 citationsDOIOpen Access PDF

Abstract

Ageing is accompanied by a decline in immune function, associated with susceptibility to infections and malignancies, and reduced vaccine efficacy. These immunological changes, affect multiple components of the immune system, particularly T lymphocytes, which exhibit altered subset distributions and accumulate senescent features. CD57, a surface glycoprotein expressed on T cells, has emerged as a potential marker of terminal differentiation and senescence used for immunomonitoring in infection or cancer contexts. However, the use of CD57 as a marker of T cell senescence remains unclear. To investigate this, we analyzed CD57 expression on CD8+ and CD4+ T cells in healthy donors from two independent cohorts, considering cellular differentiation, age, cytomegalovirus status, and other senescence markers. Our findings reinforce the association between CD57 expression, T cell differentiation, and CMV seropositivity, but not with chronological age. Although CD57 is associated with altered proliferation and survival in all T cell differentiation subsets, it does not fully align with a senescent phenotype. Therefore, we propose that CD57 may be better appreciated as a marker of immunological age. Moreover, the interpretation of CD57 expression must account for CMV serostatus to avoid misleading conclusions, especially in oncology and ageing research.

Topics & Concepts

AgeingSenescenceImmune systemImmunosenescenceBiologyT cellImmunologyCellular senescenceSerostatusCellPhenotypeT lymphocyteTelomereCellular immunityClusterinHuman cytomegalovirusNaive T cellAcquired immune systemCell growthCytotoxic T cellTelomeraseCancer researchAIM2AntibodyProliferation MarkerCell biologyCancerImmunityCTL*AntigenCytomegalovirus and herpesvirus researchNeutrophil, Myeloperoxidase and Oxidative MechanismsTelomeres, Telomerase, and Senescence
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