A plant receptor domain with functional analogies to animal malectin disables ER stress responses upon infection
Laïla Giordano, Valérie Allasia, Alexandra Cremades, Sophie Hok, Franck Panabières, Béatrice Bailly‐Maitre, Harald Keller
Abstract
mutants show decreased pathogen-responsive UPR and increased disease susceptibility. Altogether, this study revealed a previously uncharacterized role of a plant receptor domain in the regulation of ER stress during infection.
Topics & Concepts
Unfolded protein responseEndoplasmic reticulumMutantReceptorCell biologyBiologyArabidopsis thalianaPhenotypeGeneGeneticsEndoplasmic Reticulum Stress and DiseasePlant Reproductive BiologyToxin Mechanisms and Immunotoxins