TNF-α/STAT1/CXCL10 mutual inflammatory axis that contributes to the pathogenesis of experimental models of multiple sclerosis: A promising signaling pathway for targeted therapies
Roya Rahmat-Zaie, Javad Amini, Mohammad Haddadi, Cordian Beyer, Nima Sanadgol, Adib Zendedel
Topics & Concepts
PathogenesisMultiple sclerosisCXCL10MedicineSignal transductionSTAT1ImmunologyNeuroscienceBioinformaticsCancer researchBiologyInflammationChemokineCell biologyInterferonMultiple Sclerosis Research StudiesChemokine receptors and signalingNeuroinflammation and Neurodegeneration Mechanisms