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Generation of Retinal Pigment Epithelial Cells Derived from Human Embryonic Stem Cells Lacking Human Leukocyte Antigen Class I and II

Sandra Petrus-Reurer, Nerges Winblad, Pankaj Kumar, Laia Gorchs, Michael Chrobok, Arnika Kathleen Wagner, Hammurabi Bartuma, Emma Lardner, Monica Aronsson, Álvaro Plaza Reyes, Helder André, Evren Alici, Helen Kaipe, Anders Kvanta, Fredrik Lanner

2020Stem Cell Reports71 citationsDOIOpen Access PDF

Abstract

Human embryonic stem cell-derived retinal pigment epithelial (hESC-RPE) cells could serve as a replacement therapy in advanced stages of age-related macular degeneration. However, allogenic hESC-RPE transplants trigger immune rejection, supporting a strategy to evade their immune recognition. We established single-knockout beta-2 microglobulin (SKO-B2M), class II major histocompatibility complex transactivator (SKO-CIITA) and double-knockout (DKO) hESC lines that were further differentiated into corresponding hESC-RPE lines lacking either surface human leukocyte antigen class I (HLA-I) or HLA-II, or both. Activation of CD4+ and CD8+ T-cells was markedly lower by hESC-RPE DKO cells, while natural killer cell cytotoxic response was not increased. After transplantation of SKO-B2M, SKO-CIITA, or DKO hESC-RPEs in a preclinical rabbit model, donor cell rejection was reduced and delayed. In conclusion, we have developed cell lines that lack both HLA-I and -II antigens, which evoke reduced T-cell responses in vitro together with reduced rejection in a large-eyed animal model.

Topics & Concepts

BiologyCIITAEmbryonic stem cellCD8ImmunologyHuman leukocyte antigenTransplantationAntigenCell biologyMajor histocompatibility complexStem cellCytotoxic T cellImmune systemMHC class IIIn vitroGeneticsInternal medicineGeneMedicineRetinal Development and DisordersCRISPR and Genetic EngineeringNeuroinflammation and Neurodegeneration Mechanisms