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Calycosin Preserves BDNF/TrkB Signaling and Reduces Post-Stroke Neurological Injury after Cerebral Ischemia by Reducing Accumulation of Hypertrophic and TNF-α-Containing Microglia in Rats

Chien‐Chin Hsu, Ting‐Wei Kuo, Wen‐Pin Liu, Ching‐Ping Chang, Hung‐Jung Lin

2020Journal of Neuroimmune Pharmacology75 citationsDOI

Topics & Concepts

Tropomyosin receptor kinase BMicrogliaBrain-derived neurotrophic factorMedicineNeurotrophic factorsIschemiaAnesthesiaPharmacologyCalycosinNeuroprotectionInternal medicineStroke (engine)EndocrinologyReceptorInflammationFormononetinDaidzeinEngineeringGenisteinMechanical engineeringNeuroinflammation and Neurodegeneration MechanismsNerve injury and regenerationNeurogenesis and neuroplasticity mechanisms
Calycosin Preserves BDNF/TrkB Signaling and Reduces Post-Stroke Neurological Injury after Cerebral Ischemia by Reducing Accumulation of Hypertrophic and TNF-α-Containing Microglia in Rats | Litcius