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Crosstalk between PI3K/AKT/KLF4 signaling and microglia M1/M2 polarization as a novel mechanistic approach towards flibanserin repositioning in parkinson's disease

Nancy K. El-Deeb, Dalia M. El-Tanbouly, Mohamed Khattab, Mohammed F. EL-Yamany, Ahmed F. Mohamed

2022International Immunopharmacology37 citationsDOI

Topics & Concepts

NeuroprotectionMicrogliaProtein kinase BPI3K/AKT/mTOR pathwayWortmanninPharmacologyTyrosine hydroxylaseNeuroinflammationSubstantia nigraBiologyNeuroscienceSignal transductionCell biologyImmunologyDopamineInflammationDopaminergicParkinson's Disease Mechanisms and TreatmentsNeuroinflammation and Neurodegeneration MechanismsNerve injury and regeneration
Crosstalk between PI3K/AKT/KLF4 signaling and microglia M1/M2 polarization as a novel mechanistic approach towards flibanserin repositioning in parkinson's disease | Litcius