Genetic inactivation of RIP1 kinase does not ameliorate disease in a mouse model of ALS
Sara L. Domínguez, Eugene Varfolomeev, Robert P. Brendza, Kim Stark, Joy S. Tea, Jose Imperio, Hai Ngu, Timothy Earr, Oded Foreman, Joshua D. Webster, Amy Easton, Domagoj Vucic, Baris Bingol
Topics & Concepts
NecroptosisNeurodegenerationSOD1Amyotrophic lateral sclerosisKinaseTANK-binding kinase 1NeuroinflammationProtein kinase AGenetic modelBiologyGenetically modified mouseTransgeneCell biologyCancer researchMedicineNeuroscienceProgrammed cell deathMitogen-activated protein kinase kinaseInflammationImmunologyDiseasePathologyBiochemistryApoptosisGeneAmyotrophic Lateral Sclerosis ResearchAlzheimer's disease research and treatmentsAutophagy in Disease and Therapy