Galectin-3 contributes to acute cardiac dysfunction and toxicity by increasing oxidative stress and fibrosis in doxorubicin-treated mice
Ignacio M. Seropián, Florencia S. Fontana Estevez, Alejo Villaverde, Leonardo Cacciagiú, R. V. Ibáñez Bustos, Vanessa Touceda, Federico Penas, Carolina Selser, Celina Morales, Verónica Miksztowicz, Germán E. Gónzalez
Topics & Concepts
MedicineCardiotoxicityOxidative stressTBARSDoxorubicinFibrosisCardiac fibrosisInflammationLactate dehydrogenaseCardiac function curvePharmacologyInternal medicineCreatine kinaseToxicityGalectin-3Myocardial fibrosisEndocrinologyLipid peroxidationHeart failureChemotherapyBiochemistryBiologyEnzymeGalectins and Cancer BiologyChemotherapy-induced cardiotoxicity and mitigationInterstitial Lung Diseases and Idiopathic Pulmonary Fibrosis