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<i>Prdm16</i> is a critical regulator of adult long-term hematopoietic stem cell quiescence

Kristbjorn O. Gudmundsson, Nhu Nguyen, Kevin Oakley, Yufen Han, Bjorg Gudmundsdottir, Pentao Liu, Lino Tessarollo, Nancy A. Jenkins, Neal G. Copeland, Yang Du

2020Proceedings of the National Academy of Sciences35 citationsDOIOpen Access PDF

Abstract

Significance Regulation of quiescence is critical for the maintenance of adult HSCs, and the underlying mechanisms are poorly understood. Using a novel mouse conditional knockout allele of transcription factor gene Prdm16 , we show that its deletion in the adult hematopoietic system led to a gradual loss of adult HSCs over time. This loss of adult HSCs was associated with their significantly increased cycling. We further found that Prdm16 promotes the quiescence of adult HSCs by activating the transcription of HSC cell cycle inhibitors including Cdkn1a and Egr1 . Our study identifies Prdm16 as a critical regulator of adult HSC quiescence.

Topics & Concepts

HaematopoiesisBiologyRegulatorStem cellTranscription factorCell biologyHematopoietic stem cellCell cycleConditional gene knockoutPRDM16Cancer researchGeneGeneticsGene expressionPhenotypeHematopoietic Stem Cell TransplantationImmune Cell Function and InteractionT-cell and B-cell Immunology
<i>Prdm16</i> is a critical regulator of adult long-term hematopoietic stem cell quiescence | Litcius