Complement 3a induces the synapse loss via C3aR in mitochondria-dependent NLRP3 activating mechanisms during the development and progression of Alzheimer’s disease
Tong-Qi Ge, Pei-Pei Guan, Pu Wang
Topics & Concepts
SynapseNeuroscienceMitochondrionDiseaseAlzheimer's diseaseComplement (music)MedicinePsychologyBiologyCell biologyGeneticsPhenotypePathologyComplementationGeneNeuroinflammation and Neurodegeneration MechanismsAlzheimer's disease research and treatmentsTryptophan and brain disorders