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IL-17A reprograms intestinal epithelial cells to facilitate HIV-1 replication and outgrowth in CD4+ T cells

Tomas Raul Wiche Salinas, Annie Gosselin, Laurence Raymond Marchand, Etiene Moreira Gabriel, Olivier Tastet, Jean-Philippe Goulet, Yuwei Zhang, Dragos Vlad, Hanane Touil, Jean‐Pierre Routy, Mariana G. Bego, Mohamed El‐Far, Nicolas Chomont, Alan Landay, Éric A. Cohen, Cécile Tremblay, Petronela Ancuța

2021iScience13 citationsDOIOpen Access PDF

Abstract

T cells and viral outgrowth from reservoir cells of ART-treated PLWH. Importantly, the Illumina RNA-sequencing revealed an IL-17A-mediated pro-inflammatory and pro-viral molecular signature, including a decreased expression of type I interferon (IFN-I)-induced HIV restriction factors. These findings point to the deleterious features of IL-17A and raise awareness for caution when designing therapies aimed at restoring the paucity of mucosal Th17 cells in ART-treated PLWH.

Topics & Concepts

Interleukin 17ChemokineCrosstalkImmunologyCCL20Viral loadViral replicationCytokineBiologyInterferonHuman immunodeficiency virus (HIV)VirologyMedicineInflammationVirusChemokine receptorOpticsPhysicsHIV Research and TreatmentImmune Cell Function and InteractionImmunodeficiency and Autoimmune Disorders
IL-17A reprograms intestinal epithelial cells to facilitate HIV-1 replication and outgrowth in CD4+ T cells | Litcius