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Interruption of p38<sup>MAPK</sup>-MSK1-CREB-MITF-M pathway to prevent hyperpigmentation in the skin

Song-Hee Kim, Jiyeon Lee, Ji‐Hye Jung, Ga Hyun Kim, Cheong‐Yong Yun, Sang‐Hun Jung, Bang Yeon Hwang, Jin Tae Hong, Sang‐Bae Han, Jae‐Kyung Jung, Young Soo Kim

2024International Journal of Biological Sciences24 citationsDOIOpen Access PDF

Abstract

-MSK1-CREB-MITF-M pathway was suggested as a rationale to inhibit UV-B- or α-MSH-induced facultative melanogenesis and as a strategy to prevent acquired pigmentary disorders in the skin.

Topics & Concepts

Microphthalmia-associated transcription factorMelanocortin 1 receptorCREBMelanocyteTyrosinaseMelaninMelanocyte-stimulating hormoneProtein kinase ABiologyReceptorTranscription factorHairlessMAPK/ERK pathwayTLR4Molecular biologyCell biologySignal transductionEndocrinologyKinaseChemistryCancer researchBiochemistryMelanomaGenePhenotypeEnzymemelanin and skin pigmentationSkin Protection and AgingBiochemical Analysis and Sensing Techniques
Interruption of p38<sup>MAPK</sup>-MSK1-CREB-MITF-M pathway to prevent hyperpigmentation in the skin | Litcius