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Glucose homeostasis controls N-acetyltransferase 10-mediated ac4C modification of HK2 to drive gastric tumorigenesis

Qiang Wang, Mengmeng Li, Chen Chen, Lei Xu, Yao Fu, Jiawen Xu, Chuanjun Shu, Bo Wang, Zhangding Wang, Changyu Chen, Tao Song, Shouyu Wang

2025Theranostics18 citationsDOIOpen Access PDF

Abstract

Our findings highlight the critical crosstalk between glucose homeostasis and the ac4C epitranscriptome in gastric carcinogenesis. This finding offers a potential strategy of targeting NAT10/HK2 axis for the treatment of GC patients, especially those with highly active glucose metabolism.

Topics & Concepts

HomeostasisCarcinogenesisChemistryAcetyltransferaseCell biologyBiochemistryInternal medicineEndocrinologyBiologyCancer researchMedicineAcetylationGeneCancer, Hypoxia, and MetabolismAmino Acid Enzymes and MetabolismPancreatic function and diabetes
Glucose homeostasis controls N-acetyltransferase 10-mediated ac4C modification of HK2 to drive gastric tumorigenesis | Litcius