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Identification of MSA-2: An oral antitumor non-nucleotide STING agonist

Jianhua Liu, Xu Huang, Jianxun Ding

2021Signal Transduction and Targeted Therapy33 citationsDOIOpen Access PDF

Abstract

In a recent study in Science , Pan et al. 1 identified an orally available non-nucleotide human stimulator of interferon genes (STING) agonist, MSA-2 (benzothiophene oxobutanoic acid), with excellent safety tolerance in vivo. The noncovalently tethered dimers of MSA-2 could bind STING with nanomolar affinity, and the acidic tumor microenvironment would substantially increase the cell entry and retention of MSA-2 and its dimerized interaction with STING, thus leading to a superior antitumor potency of combined MSA-2 and anti-PD-1 treatments. This work has encouraged further design and discovery of clinical human STING agonists for systematic cancer therapy (Fig. 1 ). Fig. 1 Through high-throughput screening, MSA-2 was identified as a promising STING agonist from ~2.4 million compounds. Only pre-dimerized MSA-2 could bind to STING. In the acidic tumor microenvironment, uncharged MSA-2 showed a higher permeable ability and thus preferentially activated STING compared to normal tissues. Oral administration of MSA-2 exhibited excellent safety tolerance and synergetic antitumor effects with anti-PD-1 therapy in vivo Full size image

Topics & Concepts

StingStimulator of interferon genesAgonistChemistryPharmacologyImmune checkpointImmune systemInterferonGuanosineCancer immunotherapyCell cycle checkpointCancer researchT cellImmunotherapyMedicineInnate immune systemBiochemistryImmunologyReceptorCell cycleApoptosisEngineeringAerospace engineeringinterferon and immune responsesViral Infections and VectorsImmune Response and Inflammation