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Mutations in PB1, NP, HA, and NA Contribute to Increased Virus Fitness of H5N2 Highly Pathogenic Avian Influenza Virus Clade 2.3.4.4 in Chickens

Sungsu Youk, Christina M. Leyson, Brittany Seibert, Samadhan Jadhao, Daniel R. Pérez, David L. Suarez, Mary J. Pantin‐Jackwood

2020Journal of Virology26 citationsDOIOpen Access PDF

Abstract

H5Nx highly pathogenic avian influenza (HPAI) viruses of the A/goose/Guangdong/1/96 lineage continue to circulate widely affecting both poultry and wild birds. These viruses continue to change and reassort, which affects their fitness to different avian hosts. In this study, we defined mutations associated with increased virus fitness in chickens as the clade 2.3.4.4. H5N2 HPAI virus circulated in different avian species. We identified mutations in the PB1, NP, HA, and NA virus proteins that were highly conserved in the poultry isolates and contributed to the adaptation of this virus in chickens. This knowledge is important for understanding the epidemiology of H5Nx HPAI viruses and specifically the changes related to adaptation of these viruses in poultry.

Topics & Concepts

BiologyVirusVirologyInfluenza A virus subtype H5N1OutbreakInfectivityHost (biology)Influenza A virusH5N1 genetic structureGeneticsInfectious disease (medical specialty)Coronavirus disease 2019 (COVID-19)MedicinePathologyDiseaseInfluenza Virus Research StudiesAnimal Disease Management and EpidemiologyViral gastroenteritis research and epidemiology