METTL14 derived from exosomes of M1 macrophages promotes high glucose-induced apoptosis, inflammation and oxidative stress in glomerular endothelial cells by mediating PAQR3 m6A modification
Yiqun Li, Jiarong Zhang, Yanli Zhu
Topics & Concepts
MicrovesiclesOxidative stressInflammationGene knockdownFlow cytometryCell biologyMacrophageViability assayApoptosisExosomeChemistryMolecular biologyBiologyBiochemistryImmunologyIn vitromicroRNAGeneRNA modifications and cancerCancer-related gene regulationKruppel-like factors research