Litcius/Paper detail

Valeric Acid: A Gut-Derived Metabolite as a Potential Epigenetic Modulator of Neuroinflammation in the Gut–Brain Axis

Chiara Paciolla, Michele Manganelli, Mariagiovanna Di Chiano, Francesca Montenegro, Anna Gallone, Fabio Sallustio, Gabriella Guida

2025Cells18 citationsDOIOpen Access PDF

Abstract

The gut-brain axis (GBA) is a critical area of research for understanding the pathogenesis of neuroinflammatory and neurodegenerative diseases. Metabolites produced by the gut microbiota, particularly short-chain fatty acids (SCFAs), act as key mediators in this bidirectional communication. While the roles of acetate, propionate, and butyrate are well-established, valeric acid (VA), a five-carbon SCFA, is poorly understood. This comprehensive review explores VA as a gut-derived physiological epigenetic modulator, examining its microbial biosynthesis and systemic effects. This review discusses how VA acts as a selective histone deacetylase inhibitor (HDACi), particularly targeting Class I HDACs, to modulate gene expression and exert neuroprotective and anti-inflammatory effects. The analysis compares VA with its pharmacological analog, valproic acid (VPA), a well-known but non-selective HDACi. This comparison highlights how VA's physiological nature may offer a more targeted and safer intervention. In conclusion, elucidating VA's role as a microbiome-derived epigenetic regulator would open promising avenues for therapeutic strategies that directly connect gut and CNS health within the GBA.

Topics & Concepts

EpigeneticsNeuroprotectionHistone deacetylaseNeuroinflammationRegulatorValproic AcidHistoneHistone deacetylase inhibitorNeuroscienceValeric acidBiologyPharmacologyMetaboliteButyrateCell biologyChemistryKynurenine pathwayResveratrolRegulation of gene expressionEpigenesisMedicineNeurodegenerationCpG siteTrichostatin AEpigenetic therapyCRISPRCancer researchGene expressionGut microbiota and healthTryptophan and brain disordersNeuroinflammation and Neurodegeneration Mechanisms