Transitional changes in the structure of C-reactive protein create highly pro-inflammatory molecules: Therapeutic implications for cardiovascular diseases
Johannes Zeller, Balázs Bogner, James D. McFadyen, Jurij Kiefer, David Braig, Geoffrey A. Pietersz, Guy Y. Krippner, Tracy L. Nero, Craig J. Morton, Karen S. Cheung Tung Shing, Michael W. Parker, Karlheinz Peter, Steffen U. Eisenhardt
Topics & Concepts
InflammationC-reactive proteinAcute-phase proteinPathophysiologyMediatorMedicinePathologicalGene isoformMyocardial infarctionImmunologyInternal medicineChemistryBiochemistryGeneAdipokines, Inflammation, and Metabolic DiseasesInflammasome and immune disordersAtherosclerosis and Cardiovascular Diseases