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Human SLE variant NCF1-R90H promotes kidney damage and murine lupus through enhanced Tfh2 responses induced by defective efferocytosis of macrophages

Linyu Geng, Jian Zhao, Yun Deng, Ivan Molano, Xue Xu, Lingxiao Xu, Phillip Ruiz, Quan‐Zhen Li, Xuebing Feng, Miaojia Zhang, Wenfeng Tan, Diane L. Kamen, Sang‐Cheol Bae, Gary S. Gilkeson, Lingyun Sun, Betty P. Tsao

2021Annals of the Rheumatic Diseases50 citationsDOI

Topics & Concepts

Systemic lupus erythematosusMedicineImmunologyKidneyImmune systemInternal medicineEndocrinologyGerminal centerAutoantibodyEfferocytosisBiologyMacrophageAntibodyDiseaseB cellBiochemistryIn vitroNeutrophil, Myeloperoxidase and Oxidative Mechanismsinterferon and immune responsesPhagocytosis and Immune Regulation
Human SLE variant NCF1-R90H promotes kidney damage and murine lupus through enhanced Tfh2 responses induced by defective efferocytosis of macrophages | Litcius