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CCL22 mutations drive natural killer cell lymphoproliferative disease by deregulating microenvironmental crosstalk

Constance Baer, Shunsuke Kimura, Mitra S. Rana, Andrew B. Kleist, Tim Flerlage, David J. Feith, Peter Chockley, Wencke Walter, Manja Meggendorfer, Thomas L. Olson, HeeJin Cheon, Kristine C. Olson, Aakrosh Ratan, Martha-Lena Mueller, James M. Foran, Laura J. Janke, Chunxu Qu, Shaina N. Porter, Shondra M. Pruett‐Miller, Ravi Kalathur, Claudia Haferlach, Wolfgang Kern, Elisabeth Paietta, Paul G. Thomas, M. Madan Babu, Thomas P. Loughran, Ilaria Iacobucci, Torsten Haferlach, Charles G. Mullighan

2022Nature Genetics42 citationsDOIOpen Access PDF

Topics & Concepts

BiologyCCL22ChemokineChemokine receptorCrosstalkCancer researchCell biologyNatural killer cellImmunologyImmune systemGeneticsIn vitroCytotoxic T cellPhysicsOpticsImmune Cell Function and InteractionT-cell and B-cell ImmunologyLymphoma Diagnosis and Treatment
CCL22 mutations drive natural killer cell lymphoproliferative disease by deregulating microenvironmental crosstalk | Litcius