CCL22 mutations drive natural killer cell lymphoproliferative disease by deregulating microenvironmental crosstalk
Constance Baer, Shunsuke Kimura, Mitra S. Rana, Andrew B. Kleist, Tim Flerlage, David J. Feith, Peter Chockley, Wencke Walter, Manja Meggendorfer, Thomas L. Olson, HeeJin Cheon, Kristine C. Olson, Aakrosh Ratan, Martha-Lena Mueller, James M. Foran, Laura J. Janke, Chunxu Qu, Shaina N. Porter, Shondra M. Pruett‐Miller, Ravi Kalathur, Claudia Haferlach, Wolfgang Kern, Elisabeth Paietta, Paul G. Thomas, M. Madan Babu, Thomas P. Loughran, Ilaria Iacobucci, Torsten Haferlach, Charles G. Mullighan
Topics & Concepts
BiologyCCL22ChemokineChemokine receptorCrosstalkCancer researchCell biologyNatural killer cellImmunologyImmune systemGeneticsIn vitroCytotoxic T cellPhysicsOpticsImmune Cell Function and InteractionT-cell and B-cell ImmunologyLymphoma Diagnosis and Treatment