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B‐cell lymphoma/leukaemia 11B (BCL11B) expression status helps distinguish early T‐cell precursor acute lymphoblastic leukaemia/lymphoma (ETP‐ALL/LBL) from other subtypes of T‐cell ALL/LBL

Hong Fang, Wei Wang, Siba El Hussein, Kiyomi Morita, Hannah C. Beird, Akash Mitra, Sanam Loghavi, Pei Lin, Elias Jabbour, Joseph D. Khoury

2021British Journal of Haematology17 citationsDOIOpen Access PDF

Abstract

B-cell lymphoma/leukaemia 11B (BCL11B) is an essential transcription factor for T-cell lineage commitment and maturation. We investigated BCL11B expression by immunohistochemistry in T-lymphoblastic leukaemia/lymphoma (T-ALL/LBL) (n = 115). The majority (83%) of early T-cell precursor T-ALL/LBL (ETP-ALL) cases showed negative BCL11B expression, while most (84%) of non-ETP-ALL/LBL were positive for BCL11B. A simplified three-marker [BCL11B, cluster of differentiation 5 (CD5), CD13] immunophenotypic score discriminated reliably between ETP-ALL and non-ETP-ALL/LBL. In ETP-ALL, patients with positive BCL11B expression had a better overall survival than those with negative BCL11B (P = 0·009). In summary, BCL11B is a valuable marker for T-ALL/LBL subtyping and serves as a potential prognostic marker in patients with ETP-ALL.

Topics & Concepts

LymphomaImmunophenotypingT cellCD5Cancer researchB cellMedicineOncologyImmunologyMolecular biologyBiologyFlow cytometryAntibodyImmune systemAcute Lymphoblastic Leukemia researchChildhood Cancer Survivors' Quality of LifeChronic Myeloid Leukemia Treatments
B‐cell lymphoma/leukaemia 11B (BCL11B) expression status helps distinguish early T‐cell precursor acute lymphoblastic leukaemia/lymphoma (ETP‐ALL/LBL) from other subtypes of T‐cell ALL/LBL | Litcius