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HSP70-Promoter-Driven CRISPR/Cas9 System Activated by Reactive Oxygen Species for Multifaceted Anticancer Immune Response and Potentiated Immunotherapy

Liang Zhao, Dongdong Li, Yuxi Zhang, Qiaoyi Huang, Zhenghai Zhang, Chaoran Chen, Cong‐Fei Xu, Xiao Chu, Yu Zhang, Xianzhu Yang

2022ACS Nano46 citationsDOI

Abstract

To address the low response rate to immune checkpoint blockade (ICB) therapy, we propose a specific promoter-driven CRISPR/Cas9 system, F-PC/pHCP, that achieves permanent genomic disruption of PD-L1 and elicits a multifaceted anticancer immune response to potentiate immunotherapy. This system consists of a chlorin e6-encapsulated fluorinated dendrimer and HSP70-promoter-driven CRISPR/Cas9. F-PC/pHCP under 660 nm laser activated the HSP70 promoter and enabled the specific expression of the Cas9 protein to disrupt the PD-L1 gene, preventing immune escape. Moreover, F-PC/pHCP also induced immunogenic cell death (ICD) of tumor cells and reprogrammed the immunosuppressive tumor microenvironment. Overall, this specific promoter-driven CRISPR/Cas9 system showed great anticancer efficacy and, more importantly, stimulated an immune memory response to inhibit distant tumor growth and lung metastasis. This CRISPR/Cas9 system represents an alternative strategy for ICB therapy as well as enhanced cancer immunotherapy.

Topics & Concepts

CRISPRImmune systemCancer researchImmunotherapyImmune checkpointCancer immunotherapyCas9Immunogenic cell deathBiologyTumor microenvironmentImmunologyGeneGeneticsCAR-T cell therapy researchCancer Immunotherapy and BiomarkersNanoplatforms for cancer theranostics
HSP70-Promoter-Driven CRISPR/Cas9 System Activated by Reactive Oxygen Species for Multifaceted Anticancer Immune Response and Potentiated Immunotherapy | Litcius