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Inflammasome-Mediated Neuronal-Microglial Crosstalk: a Therapeutic Substrate for the Familial C9orf72 Variant of Frontotemporal Dementia/Amyotrophic Lateral Sclerosis

Kyle J. Trageser, Eun‐Jeong Yang, Chad Smith, Ruth Iban‐Arias, Tatsunori Oguchi, Maria Sebastian-Valverde, Umar Haris Iqbal, Henry Wu, Molly Estill, Md Al Rahim, Urdhva Raval, Francis Herman, Yong Jie Zhang, Leonard Petrucelli, Giulio Maria Pasinetti

2023Molecular Neurobiology18 citationsDOI

Topics & Concepts

C9orf72InflammasomeNeuroinflammationNeurodegenerationAmyotrophic lateral sclerosisFrontotemporal dementiaNeurosciencePyrin domainMicrogliaSOD1Innate immune systemBiologyMedicineDementiaImmunologyPathologyInflammationDiseaseImmune systemAmyotrophic Lateral Sclerosis ResearchAlzheimer's disease research and treatmentsNeurogenetic and Muscular Disorders Research
Inflammasome-Mediated Neuronal-Microglial Crosstalk: a Therapeutic Substrate for the Familial C9orf72 Variant of Frontotemporal Dementia/Amyotrophic Lateral Sclerosis | Litcius