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Extracellular Vesicles from <i>Lacticaseibacillus Paracasei</i> PC-H1 Inhibit HIF-1α-Mediated Glycolysis of Colon Cancer

Yangqian Shi, Chunliang Zhang, Wanyu Cao, Luyi Li, Kai‐Li Liu, Hanyue Zhu, Fikadu Balcha, Yong Fang

2024Future Microbiology19 citationsDOIOpen Access PDF

Abstract

Aims: Extracellular vesicles from Lacticaseibacillus paracasei PC-H1 have antiproliferative activity of colon cells, but the effect on glycolytic metabolism of cancer cell remains enigmatic. The authors investigated how Lacticaseibacillus paracasei extracellular vesicles (LpEVs) inhibit the growth of colon cancer cells by affecting tumor metabolism. Materials & methods: HCT116 cells were treated with LpEVs and then differentially expressed genes were analyzed by transcriptome sequencing, the sequencing results were confirmed in vivo and in vitro. Results: LpEVs entered colon cancer cells and inhibited their growth. Transcriptome sequencing revealed differentially expressed genes were related to glycolysis. Lactate production, glucose uptake and lactate dehydrogenase activity were significantly reduced after treatment. LpEVs also reduced HIF-1α, GLUT1 and LDHA expression. Conclusion: LpEVs exert their antiproliferative activity of colon cancer cells by decreasing HIF-1α-mediated glycolysis.

Topics & Concepts

GlycolysisGLUT1TranscriptomeAnaerobic glycolysisBiologyExtracellularWarburg effectLactate dehydrogenase AColorectal cancerCell biologyLactate dehydrogenaseCancer cellBiochemistryCancer researchGlucose uptakeMolecular biologyCancerMetabolismGene expressionGeneEnzymeEndocrinologyGeneticsInsulinFerroptosis and cancer prognosisRNA modifications and cancerNanoplatforms for cancer theranostics