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Bamlanivimab Treatment Leads to Rapid Selection of Immune Escape Variant Carrying the E484K Mutation in a B.1.1.7-Infected and Immunosuppressed Patient

Benedikt Lohr, Dirk Niemann, Jens Verheyen

2021Clinical Infectious Diseases40 citationsDOI

Abstract

To the Editor—Kumar et al [1] reported on the role of single bamlanivimab in preventing hospitalization associated with coronavirus disease 2019 (COVID-19). There is scarce evidence of prevalence of emergent variants in patients treated with bamlanivimab [2]. But very little is known about treatment-emergent variants in the B.1.1.7 lineage, which is by now the dominant strain throughout Europe and North America. We hereby report the selection of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) immune escape mutations in a B.1.1.7 variant 7 days after administration of single bamlanivimab in a patient with deeply impaired cellular immunity (Figure 1). The patient newly diagnosed with acute myeloid leukemia first tested positive for SARS-CoV-2 at day 11 after the start of chemotherapy. Initially, the cycle threshold (Ct) was above 30 and melting point analysis at day 15 suggested B.1.1.7 as the causative agent of this...

Topics & Concepts

MedicineImmune escapeLineage (genetic)MutationImmune systemImmunologyDiseaseMyeloid leukemiaGeneGeneticsInternal medicineBiologyImmunodeficiency and Autoimmune DisordersImmune Cell Function and InteractionSARS-CoV-2 and COVID-19 Research