Litcius/Paper detail

Trimethylamine-<i>N</i>-oxide acutely increases cardiac muscle contractility

Carlee I. Oakley, Julian Vallejo, Derek Wang, Mark Gray, LeAnn M. Tiede-Lewis, Tilitha Shawgo, Emmanuel Daon, George L. Zorn, Jason R. Stubbs, Michael Wacker

2020American Journal of Physiology-Heart and Circulatory Physiology28 citationsDOIOpen Access PDF

Abstract

We demonstrate for the first time that elevated concentrations of TMAO acutely augment myocardial contractile force ex vivo in both murine and human cardiac tissue. To gain mechanistic insight into the processes that led to this potentiation in cardiac contraction, we used two-photon microscopy to evaluate intracellular calcium in ex vivo whole hearts loaded with the calcium indicator dye Fluo-4. Acute treatment with TMAO resulted in increased Fluo-4 fluorescence, indicating that augmented cytosolic calcium plays a role in the effects of TMAO on force production. Lastly, TMAO did not show an effect on aortic smooth muscle contraction or relaxation properties. Our results demonstrate novel, acute, and direct actions of TMAO on cardiac function and help lay the groundwork for future translational studies investigating the complex multiorgan interplay involved in cardiovascular pathogenesis during CKD.

Topics & Concepts

ContractilityEx vivoContraction (grammar)CalciumChemistryCalcium in biologyMyocyteMuscle contractionLong-term potentiationCardiac function curveCytosolIn vivoInternal medicineMedicineEndocrinologyBiologyHeart failureBiochemistryIn vitroReceptorEnzymeBiotechnologyNitric Oxide and Endothelin EffectsCardiovascular Function and Risk FactorsHeart Rate Variability and Autonomic Control