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High baseline ROMK activity in the mouse late distal convoluted and early connecting tubule probably contributes to aldosterone-independent K<sup>+</sup> secretion

Viatcheslav Nesterov, Marko Bertog, Christoph Korbmacher

2021American Journal of Physiology-Renal Physiology19 citationsDOI

Abstract

ROMK-mediated renal K + secretion is essential for maintaining K + balance and requires a lumen negative transepithelial potential critically dependent on activity of the ENaC. Using microdissected distal mouse tubules, we demonstrated that baseline apical ROMK activity is high in the DCT2/CNT. Aldosterone-independent baseline ENaC activity is also high in the DCT2/CNT and downregulated by a low-K + diet, which highlights the important role of the DCT2/CNT in regulating K + secretion in an aldosterone-independent manner.

Topics & Concepts

AldosteroneEpithelial sodium channelSecretionInternal medicineEndocrinologyDistal convoluted tubuleReabsorptionChemistryHyperkalemiaPseudohypoaldosteronismMineralocorticoidKidneyBiologyMedicineSodiumOrganic chemistryIon Transport and Channel RegulationIon channel regulation and functionReceptor Mechanisms and Signaling
High baseline ROMK activity in the mouse late distal convoluted and early connecting tubule probably contributes to aldosterone-independent K<sup>+</sup> secretion | Litcius