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Hotspot DNMT3A mutations in clonal hematopoiesis and acute myeloid leukemia sensitize cells to azacytidine via viral mimicry response

Marina Scheller, Anne K. Ludwig, Stefanie Göllner, Christian Rohde, Stephen Krämer, Sina Stäble, Maike Janssen, James-Arne Müller, Lixiazi He, Nicole Bäumer, Christian Arnold, Joachim Gerß, Maximilian Schönung, Christian Thiede, Christian Niederwieser, Dietger Niederwieser, Hubert Serve, Wolfgang E. Berdel, Ulrich Thiem, Inga Hemmerling, Florian Leuschner, Christoph Plass, Matthias Schlesner, Judith B. Zaugg, Michael D. Milsom, Andreas Trumpp, Caroline Pabst, Daniel B. Lipka, Carsten Müller‐Tidow

2021Nature Cancer77 citationsDOI

Topics & Concepts

BiologyMyeloid leukemiaHaematopoiesisMyeloidCancer researchAzacitidineStem cellLeukemiaDNA methylationImmunologyGeneticsGeneGene expressionAcute Myeloid Leukemia ResearchProtein Degradation and InhibitorsEpigenetics and DNA Methylation
Hotspot DNMT3A mutations in clonal hematopoiesis and acute myeloid leukemia sensitize cells to azacytidine via viral mimicry response | Litcius