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Selenium ameliorates mercuric chloride-induced brain damage through activating BDNF/TrKB/PI3K/AKT and inhibiting NF-κB signaling pathways

Lanxin Li, Jiahong Chu, Xuewei Chen, Pei‐Chao Gao, Zhen‐Yong Wang, Ci Liu, Ruifeng Fan

2022Journal of Inorganic Biochemistry67 citationsDOI

Topics & Concepts

ChemistryNeurotoxicityProtein kinase BTropomyosin receptor kinase BSuperoxide dismutaseOxidative stressPI3K/AKT/mTOR pathwayGlutathione peroxidaseBrain-derived neurotrophic factorSignal transductionIκB kinaseNeurotrophic factorsPharmacologyEndocrinologyInternal medicineBiochemistryNF-κBReceptorToxicityBiologyMedicineOrganic chemistryMercury impact and mitigation studiesSelenium in Biological SystemsHeavy Metal Exposure and Toxicity
Selenium ameliorates mercuric chloride-induced brain damage through activating BDNF/TrKB/PI3K/AKT and inhibiting NF-κB signaling pathways | Litcius