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As a Potential Therapeutic Target, C1q Induces Synapse Loss Via Inflammasome-activating Apoptotic and Mitochondria Impairment Mechanisms in Alzheimer’s Disease

Pei-Pei Guan, Tong-Qi Ge, Pu Wang

2023Journal of Neuroimmune Pharmacology17 citationsDOI

Topics & Concepts

NeurodegenerationNeuroinflammationInflammasomeSynapseNeuroscienceMicrogliaCell biologyBiologyProinflammatory cytokineChemistryInflammationImmunologyMedicineDiseasePathologyNeuroinflammation and Neurodegeneration MechanismsAlzheimer's disease research and treatmentsInflammasome and immune disorders
As a Potential Therapeutic Target, C1q Induces Synapse Loss Via Inflammasome-activating Apoptotic and Mitochondria Impairment Mechanisms in Alzheimer’s Disease | Litcius