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Discovery of CJ-2360 as a Potent and Orally Active Inhibitor of Anaplastic Lymphoma Kinase Capable of Achieving Complete Tumor Regression

Jianyong Chen, Yunlong Zhou, Xuyuan Dong, Liu Liu, Longchuan Bai, Donna McEachern, Sally Przybranowski, Chao‐Yie Yang, Jeanne A. Stuckey, Xiaoqin Li, Bo Wen, Ting C. Zhao, Siwei Sun, Duxin Sun, Lingling Jiao, Yu Jing, Ming‐Lei Guo, Dajun Yang, Shaomeng Wang

2020Journal of Medicinal Chemistry20 citationsDOIOpen Access PDF

Abstract

We report herein the discovery of a class of potent small-molecule inhibitors of anaplastic lymphoma kinase (ALK) containing a fused indoloquinoline scaffold. The most promising compound CJ-2360 has an IC50 value of 2.2 nM against wild-type ALK and low-nanomolar potency against several clinically reported ALK mutants. This compound is capable of achieving complete tumor regression in the ALK-positive KARPAS-299 xenograft model with oral administration in mice. CJ-2360 represents a promising ALK inhibitor for advanced preclinical development.

Topics & Concepts

Anaplastic lymphoma kinaseALK inhibitorChemistryIC50PotencyKinaseLymphomaEnzyme inhibitorCancer researchPharmacologyIn vitroBiochemistryInternal medicineMedicineMalignant pleural effusionPleural effusionCancer therapeutics and mechanismsLung Cancer Treatments and MutationsLymphoma Diagnosis and Treatment