Sulforaphane ameliorates ethanol plus carbon tetrachloride-induced liver fibrosis in mice through the Nrf2-mediated antioxidant response and acetaldehyde metabolization with inhibition of the LPS/TLR4 signaling pathway
Koji Ishida, Kosuke Kaji, Shinya Sato, Hiroyuki Ogawa, Hirotetsu Takagi, Hiroaki Takaya, Hideto Kawaratani, Kei Moriya, Tadashi Namisaki, Takemi Akahane, Hitoshi Yoshiji
Topics & Concepts
SulforaphaneAcetaldehydeChemistryHepatic stellate cellHepatoprotectionAldehyde dehydrogenaseEthanol metabolismPharmacologyHepatic fibrosisLipid peroxidationCarbon tetrachlorideAlcoholic liver diseaseAntioxidantBiochemistryFibrosisEndocrinologyCancer researchInternal medicineEthanolBiologyGlutathioneMedicineCirrhosisEnzymeOrganic chemistryGeneGenomics, phytochemicals, and oxidative stressAlcohol Consumption and Health EffectsLiver Disease Diagnosis and Treatment