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Oxycodone withdrawal induces HDAC1/HDAC2-dependent transcriptional maladaptations in the reward pathway in a mouse model of peripheral nerve injury

Kerri D. Pryce, Randal A. Serafini, Aarthi Ramakrishnan, Andrew Nicolais, Ilinca M. Giosan, Claire Polizu, Angélica Torres‐Berrío, Sreeya Vuppala, Hope Kronman, Anne Ruiz, Sevasti Gaspari, Catherine J. Peña, Farhana Sakloth, Vasiliki Mitsi, John van Duzer, Ralph Mazitschek, Matthew Jarpe, Li Shen, Eric J. Nestler, Venetia Zachariou

2023Nature Neuroscience21 citationsDOIOpen Access PDF

Topics & Concepts

OxycodoneNucleus accumbensVentral tegmental areaHistone deacetylaseNeuroscienceOpioidNeuropathic painPrefrontal cortexMedicineChronic painHistone deacetylase 2μ-opioid receptorPharmacologyPsychologyBiologyInternal medicineHistoneCentral nervous systemDopamineReceptorCognitionGeneDopaminergicBiochemistryPain Mechanisms and TreatmentsPain Management and Opioid UseNeuropeptides and Animal Physiology
Oxycodone withdrawal induces HDAC1/HDAC2-dependent transcriptional maladaptations in the reward pathway in a mouse model of peripheral nerve injury | Litcius