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The antiactivator FleN uses an allosteric mechanism to regulate σ <sup>54</sup> -dependent expression of flagellar genes in <i>Pseudomonas aeruginosa</i>

Chanchal, Priyajit Banerjee, Shikha Raghav, Hemant N. Goswami, Deepti Jain

2021Science Advances36 citationsDOIOpen Access PDF

Abstract

for transcription initiation. Disruption of the conserved protein-protein interface, by mutation, shows motility and transcription defects in vivo and multiflagellate phenotype. Our study provides a detailed mechanism used by monoflagellate bacteria to fine-tune the expression of flagellar genes to form and maintain a single flagellum.

Topics & Concepts

Pseudomonas aeruginosaAllosteric regulationMechanism (biology)GeneGene expressionChemistryMicrobiologyBiologyCell biologyComputational biologyBacteriaGeneticsPhysicsReceptorQuantum mechanicsBacterial Genetics and BiotechnologyAntibiotic Resistance in BacteriaBacterial biofilms and quorum sensing
The antiactivator FleN uses an allosteric mechanism to regulate σ <sup>54</sup> -dependent expression of flagellar genes in <i>Pseudomonas aeruginosa</i> | Litcius