Litcius/Paper detail

Role of Digital Health and Artificial Intelligence in Inflammatory Bowel Disease: A Scoping Review

Kamila Majidova, Julia Handfield, Kamran Kafi, Ryan Martin, Ryszard Kubinski

2021Genes32 citationsDOIOpen Access PDF

Abstract

Inflammatory bowel diseases (IBD), subdivided into Crohn's disease (CD) and ulcerative colitis (UC), are chronic diseases that are characterized by relapsing and remitting periods of inflammation in the gastrointestinal tract. In recent years, the amount of research surrounding digital health (DH) and artificial intelligence (AI) has increased. The purpose of this scoping review is to explore this growing field of research to summarize the role of DH and AI in the diagnosis, treatment, monitoring and prognosis of IBD. A review of 21 articles revealed the impact of both AI algorithms and DH technologies; AI algorithms can improve diagnostic accuracy, assess disease activity, and predict treatment response based on data modalities such as endoscopic imaging and genetic data. In terms of DH, patients utilizing DH platforms experienced improvements in quality of life, disease literacy, treatment adherence, and medication management. In addition, DH methods can reduce the need for in-person appointments, decreasing the use of healthcare resources without compromising the standard of care. These articles demonstrate preliminary evidence of the potential of DH and AI for improving the management of IBD. However, the majority of these studies were performed in a regulated clinical environment. Therefore, further validation of these results in a real-world environment is required to assess the efficacy of these methods in the general IBD population.

Topics & Concepts

MedicineInflammatory bowel diseaseDiseaseUlcerative colitisHealth careCrohn's diseasePopulationDigital healthModalitiesQuality of life (healthcare)Intensive care medicineInternal medicineSocial scienceEnvironmental healthEconomicsEconomic growthSociologyNursingInflammatory Bowel DiseaseEosinophilic EsophagitisIL-33, ST2, and ILC Pathways